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Why this matters: GLP-1 secretion is an energy-dependent process Gut hormone-producing cells require adequate mitochondrial function Lower cellular energy can mean less efficient hormone release , including GLP-1
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The integration of all three pathways is proposed to create a complementary pharmacological profile: appetite suppression and improved insulin secretion from GLP-1 and GIP agonism, combined with enhanced energy expenditure and hepatic lipid metabolism from glucagon receptor activation
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