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Evidence ranges from early laboratory work to clinical trials depending on the use the sections below cover the actual data and sources
Colony formation assay also showed the same tendency (Fig
However, sustained activation of TLR2 and TLR4 during disease progression drives extensive neuroinflammation through microglial polarization toward pro-inflammatory M1 phenotypes and subsequent release of cytokines including TNF-, IL-6, and IL-1, which collectively activate astrocytes and accelerate neuronal death, ultimately resulting in severe neurotoxic outcomes [327]
For patients on tirzepatide, the highest-risk window is typically months 3 through 9
